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The prevailing view of people with Alzheimer’s is often a depressing one: the patient slumped in a chair or parked in front of a television set. But a new book and photo exhibition this month in New York show another side of the disease, one in which people with dementia can still be engaged, lead active lives and experience love and joy.
The book, “Love, Loss and Laughter: Seeing Alzheimer’s Differently,” was written by Cathy Greenblat, a professor emerita of sociology at Rutgers University who found a second career as a photographer. The exhibition has toured the world and is currently on display at the Michael Schimmel Center for the Arts at Pace University in Manhattan.
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Scammers bilk thousands out of the unsuspecting seniors.
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Three of the late-breaker studies (dubbed “Emerging Science” in the meeting program) address imaging agents for detecting beta-amyloid plaques in the brains of live patients.
One such product, florbetapir (Amyvid), was just approved by the FDA, but competing products are not far behind.
Meeting attendees will hear new results with two of these, florbetaben and flutemetamol. Like florbetapir, both are 18F-labeled compounds that bind selectively to beta-amyloid plaques and light up on PET scans.
The florbetaben study abstract indicates that findings on PET scans in near-death patients correlated strongly with their actual plaques measured at autopsy a short time later.
A similar study with flutemetamol came up with similar results, according to its abstract.
In a study with possibly more clinical relevance, flutemetamol binding in the brains of 11 community-dwelling older people with memory loss correlated significantly with their performance on a cognition test.
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Researchers believe they have gained key insights on the development and progression of Alzheimer’s disease.
The findings could lead to the development of antibiotic treatments that could prevent the onset of the devastating illness.
Investigators have learned that a key protein, called a tau-protein, transforms from being a critical component of normal brain function to a sinister malformed villain that destroys brain cells.
Researchers at Beth Israel Deaconess Medical Center (BIDMC) developed the technology that distinguishes the two tau isoforms — one healthy and one disease-causing. Their research shows that only the disease-causing isoform is found in the neurons of Alzheimer’s patients and is exhibited at a very early stage of disease.
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Following Alzheimer’s disease, Vascular dementia is another common cause of dementia. It is hard to believe for any individual that he is suffering from such a disease which makes him forget even his own home and family. In UK, around 200,000 people are affected by this disease. But experts still have to find out the reason behind the occurrence of this illness. The origins of vascular dementia are still unknown to them.
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